NSF award 2611174 · Brown University

Mechanisms of Base Excision Repair in Nucleosomes

Final report due

28 October 2030

in 1,471 days · The award ends on 30 June 2030.

GrantClerk is a private service. We are not NSF or any government agency.

Dates this award owes

NSF emails you about this too.

Decide about an extension2 Mar 2030in 1231 days
award ending 2030-06-30 · Research.gov
This one is ours, not NSF’s.
Extension request16 May 2030in 1306 days
award ending 2030-06-30 · Research.gov
PAPPG 24-1: “at least 45 days prior to the end date”
No-cost extension notice20 Jun 2030in 1341 days
award ending 2030-06-30 · Research.gov
PAPPG 24-1: “at least ten calendar days prior to the end date”
Final report due28 Oct 2030in 1471 days
award ending 2030-06-30 · Research.gov
PAPPG 24-1: “no later than 120-days following the end date of the award”
Project outcomes report due28 Oct 2030in 1471 days
award ending 2030-06-30 · Research.gov
PAPPG 24-1: “no later than 120 days following the end date”
Award is closed out28 Oct 2030in 1471 days
award ending 2030-06-30 · Research.gov
PAPPG 24-1: “financially closed out 120 days after their expiration date”

Annual report dates come from the budget period on your award notice. That date is not public, so it is not shown here.

How we got this date

NSF lists this award's end date as 30 June 2030.
NSF rules say the final report is due no later than 120 days after that.
So the final report is due by 28 October 2030.

Read the rule on Research.gov

Get a note before each report date

Add this award to your desk. We email you before each date and keep the dates in one place.

Renews each year until you cancel. Cancel any time from your account.

Already a member? Sign in

Award card

Award number: 2611174
Period of support: 1 Jul 2026 – 30 Jun 2030
Amount: $1,200,000
Awardee: Brown University, Providence, RI
Program: Molecular Biosciences
Directorate: BIO

Abstract

Genomic DNA in human cells is tightly packaged into chromatin, a higher‑order structure that protects the genome but also makes it difficult for essential repair enzymes to access damaged DNA. Because DNA damage threatens organismal function by promoting mutations, aging‑related decline, and disease, cells rely on the base excision repair (BER) pathway to remove and replace damaged bases. However, how BER functions…

Is this right?

We copy this from the public NSF award record. Your award notice and NSF always win. If a date looks wrong, check Research.gov.